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CEND1 Polyclonal Antibody

SKU: E-AB-19691-200

  • $ 55995


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CEND1 Polyclonal Antibody

 

SKU # E-AB-19691
Reactivity Human,  Mouse,  Rat
Host Rabbit
Applications WB,  IHC

 

Product Details

Isotype IgG
Host Rabbit
Reactivity Human,  Mouse,  Rat
Applications WB,  IHC
Clonality Polyclonal
Immunogen Synthetic peptide of human CEND1
Abbre CEND1
Synonyms BM88 ,  BM88 antigen,  Cell cycle exit and neuronal differentiation 1 ,  Cell cycle exit and neuronal differentiation protein 1,  FLJ90066,  MGC34326
Swissprot
Calculated MW 15 kDa
Observed MW Refer to figures
Cellular Localization Membrane, Single-pass type IV membrane protein.
Concentration 0.9 mg/mL
Buffer Phosphate buffered solution, pH 7.4, containing 0.05% stabilizer and 50% glycerol.
Purification Method Antigen affinity purification
Research Areas Cancer,  Cell Biology,  Neuroscience,  Tags and Cell markers
Conjugation Unconjugated
Storage Store at -20°C Valid for 12 months. Avoid freeze / thaw cycles.
Shipping The product is shipped with ice pack,upon receipt,store it immediately at the temperature recommended.

 

Related Reagents

Applications Recommended Dilution
WB 1:500-1:2000
IHC  1:40-1:200

 

Background   

BM88, also known as CEND1 (cell cycle exit and neuronal differentiation protein 1), is a 149 amino acid protein that belongs to the CEND1 familly. Involved in neuroblastoma cell differentiation, BM88 is a single-pass type IV membrane protein that is neuron specific. It is suggested that BM88 forms a dimer of two identical polypeptides linked by disulfide bridges. BM88 has a central proline-rich region containing four PxxP motifs, which typically bind SRC homology-3 (SH3) domains, as well as a putative C-terminal transmembrane region, and several potential sites for N-glycosylation, myristoylation and phosphorylation. It is also suggested that a novel signaling mechanism exists by which BM88 interferes with calcium release from inositol 1,4,5-trisphosphate-sensitive stores and exerts anti-proliferative and anti-apoptotic functions. BM88 is an important molecular target for HDAC inhibition, and transcription of BM88 is induced by trichostatin-A.