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Recombinant Mouse CXCL2/MIP-2 Protein

SKU: PKSM040996-100

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Recombinant Mouse CXCL2/MIP-2 Protein

 

SKU # PKSM040996
Expression Host E.coli

 

 

Description

Synonyms C-X-C motif chemokine 2, GRO beta, GRO2, GROB, Gro-beta, Growth-regulated protein beta, MGSA-b, MGSA-beta, MIP-2, MIP2-alpha, MIP2A, Macrophage Inflammatory Protein-2-alpha, SCYB2., chemokine ligand 2, cxcl2, melanoma growth stimulatory activity beta
Species Mouse
Expression Host E.coli
Sequence Ala28-Asn100
Accession P10889
Calculated Molecular Weight 8.7 kDa
Observed Molecular Weight 11 kDa
Tag N-His
Bio-activity Measure by its ability to chemoattract BaF3 cells transfected with human CXCR2.The ED50 for this effect is <0.5 ng/mL.
  

 

Properties

Purity > 98 % as determined by reducing SDS-PAGE.
Endotoxin < 0.1 EU per μg of the protein as determined by the LAL method.
Storage Generally, lyophilized proteins are stable for up to 12 months when stored at -20 to -80℃. Reconstituted protein solution can be stored at 4-8℃ for 2-7 days. Aliquots of reconstituted samples are stable at < -20℃ for 3 months.
Shipping This product is provided as lyophilized powder which is shipped with ice packs.
Formulation Lyophilized from sterile PBS, pH 7.4.
Normally 5% - 8% trehalose, mannitol and 0.01% Tween 80 are added as protectants before lyophilization.
Please refer to the specific buffer information in the printed manual.
Reconstitution Please refer to the printed manual for detailed information.



Background

C-X-C motif chemokine 2 (CXCL2,MIP-2) belongs to the intercrine alpha (chemokine CxC) family. It was originally identified as a heparin-binding protein secreted from a murine macrophage cell line in response to endotoxin stimulation. The expression of mouse MIP-2 is stimulated by endotoxin. The mouse MIP-2 shares approximately 63% aa sequence identity with murine KC, another mouse alpha chemokine, which is induced by PDGF. It has been suggested that mouse KC and MIP-2 are the homologs of the human GROs and rat CINCs. Chemotactic for human polymorphonuclear leukocytes but does not induce chemokinesis or an oxidative burst. The expression of MIP-2 was found to be associated with neutrophil influx in pulmonary inflammation and glomerulonephritis, suggesting that MIP-2 may contribute to the pathogenesis of inflammatory diseases.